Post-alcoholic hypoglycaemia: a clinical and pathological study

dc.contributor.authorNaeme, Peter Brereton
dc.date.accessioned2020-06-02T11:17:13Z
dc.date.available2020-06-02T11:17:13Z
dc.date.issued1965
dc.date.updated2020-04-09T10:28:32Z
dc.description.abstractFor a number of years a history of sudden onset of coma, followed later by death, was noted amongst Africans at King Edward VIII hospital, Durban. Necropsy, where performed, revealed no cause in many of these patients. In 1957 after cerebrospinal fluid had been examined in some of the cases it was seen that the only abnormality present was a low cerebrospinal sugar. Hypoglycaemia was suspected and blood sugar estimation confirmed it. Further investigation failed to reveal the cause of the hypoglycaemia and in some cases because of a "flat" glucose tolerance curve organic hyperinsulinism was suspected. In one case total pancreatectomy was performed but thorough macroscopical and histological investigation failed to reveal a tumour or hyperplasia, and the patient subsequently suffered from diabetes. In August 1958, I admitted an African female patient, of 26 years, to the ward in coma.
dc.identifier.apacitationNaeme, P. B. (1965). <i>Post-alcoholic hypoglycaemia: a clinical and pathological study</i>. (). ,Faculty of Health Sciences ,Department of Medicine. Retrieved from en_ZA
dc.identifier.chicagocitationNaeme, Peter Brereton. <i>"Post-alcoholic hypoglycaemia: a clinical and pathological study."</i> ., ,Faculty of Health Sciences ,Department of Medicine, 1965. en_ZA
dc.identifier.citationNaeme, P.B. 1965. Post-alcoholic hypoglycaemia: a clinical and pathological study. . ,Faculty of Health Sciences ,Department of Medicine. en_ZA
dc.identifier.ris TY - Thesis / Dissertation AU - Naeme, Peter Brereton AB - For a number of years a history of sudden onset of coma, followed later by death, was noted amongst Africans at King Edward VIII hospital, Durban. Necropsy, where performed, revealed no cause in many of these patients. In 1957 after cerebrospinal fluid had been examined in some of the cases it was seen that the only abnormality present was a low cerebrospinal sugar. Hypoglycaemia was suspected and blood sugar estimation confirmed it. Further investigation failed to reveal the cause of the hypoglycaemia and in some cases because of a "flat" glucose tolerance curve organic hyperinsulinism was suspected. In one case total pancreatectomy was performed but thorough macroscopical and histological investigation failed to reveal a tumour or hyperplasia, and the patient subsequently suffered from diabetes. In August 1958, I admitted an African female patient, of 26 years, to the ward in coma. DA - 1965 DB - OpenUCT DP - University of Cape Town KW - Alcoholism KW - Complications LK - https://open.uct.ac.za PY - 1965 T1 - Post-alcoholic hypoglycaemia: a clinical and pathological study TI - Post-alcoholic hypoglycaemia: a clinical and pathological study UR - ER - en_ZA
dc.identifier.urihttps://hdl.handle.net/11427/32024
dc.identifier.vancouvercitationNaeme PB. Post-alcoholic hypoglycaemia: a clinical and pathological study. []. ,Faculty of Health Sciences ,Department of Medicine, 1965 [cited yyyy month dd]. Available from: en_ZA
dc.language.rfc3066eng
dc.publisher.departmentDepartment of Medicine
dc.publisher.facultyFaculty of Health Sciences
dc.subjectAlcoholism
dc.subjectComplications
dc.titlePost-alcoholic hypoglycaemia: a clinical and pathological study
dc.typeDoctoral Thesis
dc.type.qualificationlevelDoctoral
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