Arteannuin-B and (3-Chlorophenyl)-2-Spiroisoxazoline Derivative Exhibit Anti-Inflammatory Effects in LPS-Activated RAW 264.7 Macrophages and BALB/c Mice-Induced Proinflammatory Responses via Downregulation of NF-κB/P38 MAPK Signaling

dc.contributor.authorSawhney, Gifty
dc.contributor.authorRasool, Javeed Ur
dc.contributor.authorSaroch, Diksha
dc.contributor.authorOzturk, Mumin
dc.contributor.authorBrombacher, Frank
dc.contributor.authorAhmad, Bilal
dc.contributor.authorBhagat, Asha
dc.contributor.authorAli, Asif
dc.contributor.authorParihar, Suraj P.
dc.contributor.authorAhmed, Zabeer
dc.date.accessioned2024-04-29T10:29:59Z
dc.date.available2024-04-29T10:29:59Z
dc.date.issued2022-11-20
dc.date.updated2022-11-24T14:43:28Z
dc.description.abstractHost inflammatory responses are key to protection against injury; however, persistent inflammation is detrimental and contributes to morbidity and mortality. Herein, we demonstrated the anti-inflammatory role of Arteannuin-B (<b>1</b>) and its new spirocyclic-2-isoxazoline derivative <b>JR-9</b> and their side effects in acute inflammatory condition in vivo using LPS-induced cytokines assay, carrageenan-induced paw edema, acetic acid-induced writhing and tail immersion. The results show that the spirocyclic-2-isoxazoline derivative is a potent anti-inflammatory agent with minimal cell toxicity as compared to Arteannuin-B. In addition, the efficacies of these compounds were also validated by flow cytometric, computational, and histopathological analysis. Our results show that the anti-inflammatory response of <b>JR-9</b> significantly reduces the ability of mouse macrophages to produce NO, TNF-&alpha;, and IL-6 following LPS stimulation. Therefore, JR-9 is a prospective candidate for the development of anti-inflammatory drugs and its molecular mechanism is likely related to the regulation of NF-&kappa;B and MAPK signaling pathway.
dc.identifierdoi: 10.3390/molecules27228068
dc.identifier.apacitationSawhney, G., Rasool, J. U., Saroch, D., Ozturk, M., Brombacher, F., Ahmad, B., ... Ahmed, Z. (2022). Arteannuin-B and (3-Chlorophenyl)-2-Spiroisoxazoline Derivative Exhibit Anti-Inflammatory Effects in LPS-Activated RAW 264.7 Macrophages and BALB/c Mice-Induced Proinflammatory Responses via Downregulation of NF-&kappa;B/P38 MAPK Signaling. http://hdl.handle.net/11427/39483en_ZA
dc.identifier.chicagocitationSawhney, Gifty, Javeed Ur Rasool, Diksha Saroch, Mumin Ozturk, Frank Brombacher, Bilal Ahmad, Asha Bhagat, Asif Ali, Suraj P. Parihar, and Zabeer Ahmed "Arteannuin-B and (3-Chlorophenyl)-2-Spiroisoxazoline Derivative Exhibit Anti-Inflammatory Effects in LPS-Activated RAW 264.7 Macrophages and BALB/c Mice-Induced Proinflammatory Responses via Downregulation of NF-&kappa;B/P38 MAPK Signaling." (2022) http://hdl.handle.net/11427/39483en_ZA
dc.identifier.citationMolecules 27 (22): 8068 (2022)
dc.identifier.ris TY - Journal Article AU - Sawhney, Gifty AU - Rasool, Javeed Ur AU - Saroch, Diksha AU - Ozturk, Mumin AU - Brombacher, Frank AU - Ahmad, Bilal AU - Bhagat, Asha AU - Ali, Asif AU - Parihar, Suraj P. AU - Ahmed, Zabeer AB - Host inflammatory responses are key to protection against injury; however, persistent inflammation is detrimental and contributes to morbidity and mortality. Herein, we demonstrated the anti-inflammatory role of Arteannuin-B (<b>1</b>) and its new spirocyclic-2-isoxazoline derivative <b>JR-9</b> and their side effects in acute inflammatory condition in vivo using LPS-induced cytokines assay, carrageenan-induced paw edema, acetic acid-induced writhing and tail immersion. The results show that the spirocyclic-2-isoxazoline derivative is a potent anti-inflammatory agent with minimal cell toxicity as compared to Arteannuin-B. In addition, the efficacies of these compounds were also validated by flow cytometric, computational, and histopathological analysis. Our results show that the anti-inflammatory response of <b>JR-9</b> significantly reduces the ability of mouse macrophages to produce NO, TNF-&alpha;, and IL-6 following LPS stimulation. Therefore, JR-9 is a prospective candidate for the development of anti-inflammatory drugs and its molecular mechanism is likely related to the regulation of NF-&kappa;B and MAPK signaling pathway. DA - 2022-11-20 DB - OpenUCT DP - University of Cape Town LK - https://open.uct.ac.za PY - 2022 T1 - Arteannuin-B and (3-Chlorophenyl)-2-Spiroisoxazoline Derivative Exhibit Anti-Inflammatory Effects in LPS-Activated RAW 264.7 Macrophages and BALB/c Mice-Induced Proinflammatory Responses via Downregulation of NF-&kappa;B/P38 MAPK Signaling TI - Arteannuin-B and (3-Chlorophenyl)-2-Spiroisoxazoline Derivative Exhibit Anti-Inflammatory Effects in LPS-Activated RAW 264.7 Macrophages and BALB/c Mice-Induced Proinflammatory Responses via Downregulation of NF-&kappa;B/P38 MAPK Signaling UR - http://hdl.handle.net/11427/39483 ER - en_ZA
dc.identifier.urihttp://hdl.handle.net/11427/39483
dc.identifier.vancouvercitationSawhney G, Rasool JU, Saroch D, Ozturk M, Brombacher F, Ahmad B, et al. Arteannuin-B and (3-Chlorophenyl)-2-Spiroisoxazoline Derivative Exhibit Anti-Inflammatory Effects in LPS-Activated RAW 264.7 Macrophages and BALB/c Mice-Induced Proinflammatory Responses via Downregulation of NF-&kappa;B/P38 MAPK Signaling. 2022; http://hdl.handle.net/11427/39483.en_ZA
dc.titleArteannuin-B and (3-Chlorophenyl)-2-Spiroisoxazoline Derivative Exhibit Anti-Inflammatory Effects in LPS-Activated RAW 264.7 Macrophages and BALB/c Mice-Induced Proinflammatory Responses via Downregulation of NF-&kappa;B/P38 MAPK Signaling
dc.typeJournal Article
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