The crystal structure of halofantrine–ferriprotoporphyrin IX and the mechanism of action of arylmethanol antimalarials.

dc.contributor.authorde Villiers, Katherine A
dc.contributor.authorMarques, Helder
dc.contributor.authorEgan, Timothy J
dc.date.accessioned2016-07-20T13:37:22Z
dc.date.available2016-07-20T13:37:22Z
dc.date.issued2008
dc.date.updated2016-07-20T13:36:10Z
dc.description.abstractThe crystal structure of the complex formed between the antimalarial drug halofantrine and ferriprotoporphyrin IX (Fe(III)PPIX) has been determined by single crystal X-ray diffraction. The structure shows that halofantrine coordinates to the Fe(III) center through its alcohol functionality in addition to p-stacking of the phenanthrene ring over the porphyrin. The length of the Fe(III)–O bond is consistent with an alkoxide and not an alcohol coordinating group. The iron porphyrin is five coordinate and monomeric. Changes in the electronic spectrum of Fe(III)PPIX upon addition of halofantrine base in acetonitrile solution are almost identical to those observed upon addition of quinidine free base in the same solvent. This suggests homologous binding. Molecular mechanics modeling of Fe(III)PPIX complexes of quinidine, quinine, 9-epiquinine and 9-epiquinidine based on this homology suggests that the antimalarially active quinidine and quinine can readily adopt conformations that permit formation of an intramolecular salt bridge between the protonated quinuclidine tertiary amino group and unprotonated heme propionate group, while the inactive epimers 9-epiquinidine and 9-epiquinine have to adopt high energy conformations in order to accommodate such salt bridge formation. We propose that salt bridge formation may interrupt formation of the hemozoin precursor dimer formed during the heme detoxification pathway and so account for the strong activity of the two active isomers.en_ZA
dc.identifierhttp://dx.doi.org/10.1016/j.jinorgbio.2008.04.001
dc.identifier.apacitationde Villiers, K. A., Marques, H., & Egan, T. J. (2008). The crystal structure of halofantrine–ferriprotoporphyrin IX and the mechanism of action of arylmethanol antimalarials. <i>Journal of Inorganic Biochemistry</i>, http://hdl.handle.net/11427/20552en_ZA
dc.identifier.chicagocitationde Villiers, Katherine A, Helder Marques, and Timothy J Egan "The crystal structure of halofantrine–ferriprotoporphyrin IX and the mechanism of action of arylmethanol antimalarials." <i>Journal of Inorganic Biochemistry</i> (2008) http://hdl.handle.net/11427/20552en_ZA
dc.identifier.citationde Villiers, K. A., Marques, H. M., & Egan, T. J. (2008). The crystal structure of halofantrine–ferriprotoporphyrin IX and the mechanism of action of arylmethanol antimalarials. Journal of inorganic biochemistry, 102(8), 1660-1667.en_ZA
dc.identifier.issn0162-0134en_ZA
dc.identifier.ris TY - Journal Article AU - de Villiers, Katherine A AU - Marques, Helder AU - Egan, Timothy J AB - The crystal structure of the complex formed between the antimalarial drug halofantrine and ferriprotoporphyrin IX (Fe(III)PPIX) has been determined by single crystal X-ray diffraction. The structure shows that halofantrine coordinates to the Fe(III) center through its alcohol functionality in addition to p-stacking of the phenanthrene ring over the porphyrin. The length of the Fe(III)–O bond is consistent with an alkoxide and not an alcohol coordinating group. The iron porphyrin is five coordinate and monomeric. Changes in the electronic spectrum of Fe(III)PPIX upon addition of halofantrine base in acetonitrile solution are almost identical to those observed upon addition of quinidine free base in the same solvent. This suggests homologous binding. Molecular mechanics modeling of Fe(III)PPIX complexes of quinidine, quinine, 9-epiquinine and 9-epiquinidine based on this homology suggests that the antimalarially active quinidine and quinine can readily adopt conformations that permit formation of an intramolecular salt bridge between the protonated quinuclidine tertiary amino group and unprotonated heme propionate group, while the inactive epimers 9-epiquinidine and 9-epiquinine have to adopt high energy conformations in order to accommodate such salt bridge formation. We propose that salt bridge formation may interrupt formation of the hemozoin precursor dimer formed during the heme detoxification pathway and so account for the strong activity of the two active isomers. DA - 2008 DB - OpenUCT DP - University of Cape Town J1 - Journal of Inorganic Biochemistry LK - https://open.uct.ac.za PB - University of Cape Town PY - 2008 SM - 0162-0134 T1 - The crystal structure of halofantrine–ferriprotoporphyrin IX and the mechanism of action of arylmethanol antimalarials TI - The crystal structure of halofantrine–ferriprotoporphyrin IX and the mechanism of action of arylmethanol antimalarials UR - http://hdl.handle.net/11427/20552 ER - en_ZA
dc.identifier.urihttp://hdl.handle.net/11427/20552
dc.identifier.urihttp://www.sciencedirect.com/science/article/pii/S0162013408001141
dc.identifier.vancouvercitationde Villiers KA, Marques H, Egan TJ. The crystal structure of halofantrine–ferriprotoporphyrin IX and the mechanism of action of arylmethanol antimalarials. Journal of Inorganic Biochemistry. 2008; http://hdl.handle.net/11427/20552.en_ZA
dc.languageengen_ZA
dc.publisherElsevieren_ZA
dc.publisher.institutionUniversity of Cape Town
dc.sourceJournal of Inorganic Biochemistryen_ZA
dc.source.urihttp://www.journals.elsevier.com/journal-of-inorganic-biochemistry/
dc.subject.otherMalaria
dc.subject.otherAntimalarials
dc.subject.otherHalofantrine
dc.subject.otherQuinine
dc.subject.otherQuinidine
dc.subject.otherHeme
dc.subject.otherCrystal structure
dc.titleThe crystal structure of halofantrine–ferriprotoporphyrin IX and the mechanism of action of arylmethanol antimalarials.en_ZA
dc.typeJournal Articleen_ZA
uct.type.filetypeText
uct.type.filetypeImage
uct.type.publicationResearchen_ZA
uct.type.resourceArticleen_ZA
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